AddexBio BEAS-2B cells (来源于支气管上皮) BEAS-2B cells(T0007001)

Origin: Bronchus epithelial, cells were isolated from normal human bronchial epithelium obtained from autopsy of non-cancerous individuals. Species:Homo sapiens Tissue:Lung, Bronchus Morphology:Epithelial Properties:Adherent Cytogenic data:Normal, SV40 immortalized, and Non-tumorigenic Medium:BEGM + additives Subculture:when confluent using 0.25% trypsin or trypsin/EDTA, 5% CO2; 37 °C Freezing Medium:Complete culture medium supplemented with 5% (v/v) DMSO Biosafety Level:II Sterility:Bacteria: Negative Yeast: Negative Mycoplasma: Negative Pathogens:HIV: Negative Hepatitis B: Negative Hepatitis C: NegativeReferences for BEAS-2B cells:1. Fuentes-Mattei, E., Rivera, E., Gioda, A., Sanchez-Rivera, D., Roman-Velazquez, F. R., and Jimenez-Velez, B. D. Use of human bronchial epithelial cells (BEAS-2B) to study immunological markers resulting from exposure to PM(2.5) organic extract from Puerto Rico. Toxicol Appl Pharmacol, 243: 381-389. 2. Chilakapati, J., Wallace, K., Ren, H., Fricke, M., Bailey, K., Ward, W., Creed, J., and Kitchin, K. Genome-wide analysis of BEAS-2B cells exposed to trivalent arsenicals and dimethylthioarsinic acid. Toxicology, 268: 31-39. 3. Ovrevik, J., Arlt, V. M., Oya, E., Nagy, E., Mollerup, S., Phillips, D. H., Lag, M., and Holme, J. A. Differential effects of nitro-PAHs and amino-PAHs on cytokine and chemokine responses in human bronchial epithelial BEAS-2B cells. Toxicol Appl Pharmacol, 242: 270-280. 4. Antognelli, C., Gambelunghe, A., Del Buono, C., Murgia, N., Talesa, V. N., and Muzi, G. Crystalline silica Min-U-Sil 5 induces oxidative stress in human bronchial epithelial cells BEAS-2B by reducing the efficiency of antiglycation and antioxidant enzymatic defenses. Chem Biol Interact, 182: 13-21, 2009. 5. Eom, H. J. and Choi, J. Oxidative stress of silica nanoparticles in human bronchial epithelial cell, BEAS-2B. Toxicol In Vitro, 23: 1326-1332, 2009. 6. Eom, H. J. and Choi, J. Oxidative stress of CeO2 nanoparticles via p38-Nrf-2 signaling pathway in human bronchial epithelial cell, BEAS-2B. Toxicol Lett, 187: 77-83, 2009. 7. Li, Q., Suen, T. C., Sun, H., Arita, A., and Costa, M. Nickel compounds induce apoptosis in human bronchial epithelial BEAS-2B cells by activation of c-Myc through ERK pathway. Toxicol Appl Pharmacol, 235: 191-198, 2009. 8. Park, E. J. and Park, K. Induction of pro-inflammatory signals by 1-nitropyrene in cultured BEAS-2B cells. Toxicol Lett, 184: 126-133, 2009.